Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Association of the T102C polymorphism in the HTR2A gene with major depressive disorder, bipolar disorder, and schizophrenia.

American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics - 1 Jul 2014

Tan Jinjing, Chen Shan, Su Li, Long Jianxiong, Xie Juanjuan, Shen Tingting, Jiang Juan, Gu Lian

Abstract excerpt

A number of studies have assessed a relationship between the T102C polymorphism in the HTR2A gene with an increased risk of major depressive disorder (MDD), bipolar disorder (BPD), and schizophrenia (SCZ). However, the results have been inconsistent. Hence, we performed this study to further evaluate potential associations between the T102C polymorphism and MDD, BPD, and SCZ. The strength of separate associations...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Asian People
Share to Bipolar Disorder

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Depressive Disorder, Major

Share to Genetic Predisposition to Disease

Share to Genotype

Share to Polymorphism, Genetic

Share to Receptor, Serotonin, 5-HT2A

Share to Schizophrenia

Source

Identifiers

PMID: 24962835
DOI: 10.1002/ajmg.b.32248

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Association of the T102C polymorphism in the HTR2A gene with major depressive disorder, bipolar disorder, and schizophrenia.PMID 24962835Read ↗

Association of HTR2A T102C and A-1438G polymorphisms with susceptibility to major depressive disorder: a meta-analysis.Semantic proximity Read ↗

HTR2A-1438A/G polymorphism influences the risk of schizophrenia but not bipolar disorder or major depressive disorder: a meta-analysis.Semantic proximity Read ↗

Association between the T102C polymorphism of the serotonin-2A receptor gene and schizophrenia.Semantic proximity Read ↗

Hypomethylation of the serotonin receptor type-2A Gene (HTR2A) at T102C polymorphic site in DNA derived from the saliva of patients with schizophrenia and bipolar disorder.Semantic proximity Read ↗

Genetic association analysis of serotonin 2A receptor gene (HTR2A) with bipolar disorder and major depressive disorder in the Japanese population.Semantic proximity Read ↗

Negative association between T102C polymorphism of the 5-HT2a receptor gene and schizophrenia in Japan.Semantic proximity Read ↗

Supportive evidence for the association between the T102C 5-HTR2A gene polymorphism and schizophrenia: a large-scale case-control and family-based study.Semantic proximity Read ↗

Meta-analysis of association between the T102C polymorphism of the 5HT2a receptor gene and schizophrenia.Semantic proximity Read ↗

Epigenetic dysregulation of HTR2A in the brain of patients with schizophrenia and bipolar disorder.Semantic proximity Read ↗

Association of the 5HTR2A gene with suicidal behavior: case-control study and updated meta-analysis.Semantic proximity Read ↗

Association between HTR2C gene variants and suicidal behaviour: a protocol for the systematic review and meta-analysis of genetic studies.Semantic proximity Read ↗

Genetic Association of HTR1B and HTR2A Gene Polymorphisms with ADHD in Korean Children and Adolescents: A Case Control Study.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.