Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Evolution of hypervirulence by a MRSA clone through acquisition of a transposable element

24 Jun 2014

Abstract excerpt

Staphylococcus aureus has evolved as a pathogen that causes a range of diseases in humans. There are two dominant modes of evolution thought to explain most of the virulence differences between strains. First, virulence genes may be acquired from other organisms. Second, mutations may cause changes in the regulation and expression of genes. Here we describe an evolutionary event in which transposition of an IS...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Antimicrobial Resistance in Staphylococcus
Share to Bacterial Genetics and Biotechnology
Share to Bacteriophages and microbial interactions

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 24962815
DOI: 10.1111/mmi.12682

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Evolution of hypervirulence by a MRSA clone through acquisition of a transposable elementPMID 24962815Read ↗

A potential role of transposon IS431 in the loss of mecA gene.Semantic proximity Read ↗

Increase in IS256 transposition in invasive vancomycin heteroresistant Staphylococcus aureus isolate belonging to ST100 and its derived VISA mutants.Semantic proximity Read ↗

Influence of IS256 on Genome Variability and Formation of Small-Colony Variants in Staphylococcus aureus.Semantic proximity Read ↗

Hyperexpression of α-hemolysin explains enhanced virulence of sequence type 93 community-associated methicillin-resistant Staphylococcus aureusSemantic proximity Read ↗

Targeted IS-element sequencing uncovers transposition dynamics during selective pressure in enterococciSemantic proximity Read ↗

Targeted IS-element sequencing uncovers transposition dynamics during selective pressure in enterococciSemantic proximity Read ↗

The Role of the Staphylococcal VraTSR Regulatory System on Vancomycin Resistance and vanA Operon Expression in Vancomycin-Resistant Staphylococcus aureusSemantic proximity Read ↗

Autoregulation and Virulence Control by the Toxin-Antitoxin System SavRS in Staphylococcus aureusSemantic proximity Read ↗

Decreased Vancomycin Susceptibility in Staphylococcus aureus Caused by IS <i>256</i> Tempering of WalKR ExpressionSemantic proximity Read ↗

Screening for Highly Transduced Genes in Staphylococcus aureus Revealed Both Lateral and Specialized TransductionSemantic proximity Read ↗

Influence of ciprofloxacin and vancomycin on mutation rate and transposition of IS256 in Staphylococcus aureus.Semantic proximity Read ↗

The gene for toxic shock toxin is carried by a family of mobile pathogenicity islands in Staphylococcus aureus.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.