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Article

IL-17 protects T cells from apoptosis and contributes to development of ALPS-like phenotypes.

Blood - 20 Feb 2014

Boggio Elena, Clemente Nausicaa, Mondino Anna, Cappellano Giuseppe, Orilieri Elisabetta, Gigliotti Casimiro L, Toth Erika, Ramenghi Ugo, Dianzani Umberto, Chiocchetti Annalisa

Abstract excerpt

In autoimmune/lymphoproliferative syndrome (ALPS), defective Fas death receptor function causes lymphadenomegaly/splenomegaly, the expansion of T-cell receptor αβ(+) CD4/CD8 double-negative T cells, and frequent development of hematologic autoimmunity. Dianzani autoimmune lymphoproliferative dise...

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Share to Receptors, Antigen, T-Cell, alpha-beta

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Source

Identifiers

PMID: 24363402
DOI: 10.1182/blood-2013-07-518167

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026. Prepared record publication-24363402-2026-09-04.2. Manifest publication-graph-cohort-2026-09-04.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 4 Sept 2026.

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