Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Deficiency in p53 is required for doxorubicin induced transcriptional activation of NF-κB target genes in human breast cancer

23 Nov 2013

Abstract excerpt

// Alba Dalmases 1,2 , Irene González 1,2 , Silvia Menendez 1,2 , Oriol Arpí 1,2 , Josep Maria Corominas 3 , Sonia Servitja 1,2 , Ignasi Tusquets 1,2,4 , Cristina Chamizo 5 , Raúl Rincón 5 , Lluis Espinosa 1 , Anna Bigas 1 , Pilar Eroles 6 , Jessica Furriol 6 , Anna Lluch 7,8 , Ana Rovira 1,2 , Joan Albanell 1,2,9 , Federico Rojo 1,5 1 Cancer Research Program, IMIM (Hospital del Mar Research Institute),...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer-related Molecular Pathways
Share to Metastasis and carcinoma case studies
Share to NF-κB Signaling Pathways

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 24344116
DOI: 10.18632/oncotarget.1556

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 1 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Deficiency in p53 is required for doxorubicin induced transcriptional activation of NF-κB target genes in human breast cancerPMID 24344116Read ↗

Selective activation of NF-κB subunits in human breast cancer: potential roles for NF-κB2/p52 and for Bcl-3Semantic proximity Read ↗

Inhibition of the Canonical IKK/NF&kappa;B Pathway Sensitizes Human Cancer Cells to DoxorubicinSemantic proximity Read ↗

Flavopiridol inhibits FOXM1/NF-kB and KRAS/MAPK signaling and DNA repair mechanims and Sensitizes Triple-Negative Breast Cancer Cells to Doxorubicin theSemantic proximity Read ↗

The IL-8/NF-κB feedback loop confers a paclitaxel-sensitive/doxorubicin-resistant phenotype in triple-negative breast cancerSemantic proximity Read ↗

Can NF-κB Be Considered a Valid Drug Target in Neoplastic Diseases? Our Point of ViewSemantic proximity Read ↗

A BRCA1 deficient, NFκB driven immune signal predicts good outcome in triple negative breast cancer.Semantic proximity Read ↗

p53 in breast cancer subtypes and new insights into response to chemotherapy.Semantic proximity Read ↗

FOXM1 mediates Dox resistance in breast cancer by enhancing DNA repairSemantic proximity Read ↗

PUMA and NF-kB Are Cell Signaling Predictors of Reovirus Oncolysis of Breast CancerSemantic proximity Read ↗

Histone deacetylase 2 and N-Myc reduce p53 protein phosphorylation at serine 46 by repressing gene transcription of tumor protein 53-induced nuclear protein 1Semantic proximity Read ↗

Expression of nuclear XIAP associates with cell growth and drug resistance and confers poor prognosis in breast cancer.Semantic proximity Read ↗

DDX58 deficiency leads to doxorubicin chemotherapy resistance by hindering Type I IFN signalling-mediated apoptosis in TNBCSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.