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Article

Nav 1.5 mutations linked to dilated cardiomyopathy phenotypes: Is the gating pore current the missing link?

Channels (Austin, Tex.) - 1 Jan 2014

Gosselin-Badaroudine Pascal, Moreau Adrien, Chahine Mohamed

Abstract excerpt

Nav 1.5 dysfunctions are commonly linked to rhythms disturbances that include type 3 long QT syndrome (LQT3), Brugada syndrome (BrS), sick sinus syndrome (SSS) and conduction defects. Recently, this channel protein has been also linked to structural heart diseases such as dilated cardiomyopathy (DCM).

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Share to Cardiac electrophysiology and arrhythmias
Share to Cardiomyopathy, Dilated
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Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

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Source

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Identifiers

PMID: 24300601
DOI: 10.4161/chan.27179
PMCID: PMC4048347

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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