Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The BH3-only SNARE BNip1 mediates photoreceptor apoptosis in response to vesicular fusion defects.

Developmental cell - 28 May 2013

Nishiwaki Yuko, Yoshizawa Asuka, Kojima Yutaka, Oguri Eri, Nakamura Shohei, Suzuki Shohei, Yuasa-Kawada Junichi, Kinoshita-Kawada Mariko, Mochizuki Toshiaki, Masai Ichiro

Abstract excerpt

Intracellular vesicular transport is important for photoreceptor function and maintenance. However, the mechanism underlying photoreceptor degeneration in response to vesicular transport defects is unknown. Here, we report that photoreceptors undergo apoptosis in a zebrafish β-soluble N-ethylmaleimide-sensitive factor attachment protein (β-SNAP) mutant. β-SNAP cooperates with N-ethylmaleimide-sensitive factor to...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Endoplasmic Reticulum
Share to Golgi Apparatus

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Membrane Fusion

Share to Multiprotein Complexes

Share to Protein Interaction Mapping

Share to Proto-Oncogene Proteins c-bcl-2

Share to Qa-SNARE Proteins

Share to Retinal Cone Photoreceptor Cells

Source

Identifiers

PMID: 23725763
DOI: 10.1016/j.devcel.2013.04.015

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The BH3-only SNARE BNip1 mediates photoreceptor apoptosis in response to vesicular fusion defects.PMID 23725763Read ↗

Neuronal necrosis and spreading death in a Drosophila genetic modelSemantic proximity Read ↗

Syntaxin 3B: A SNARE Protein Required for Vision.Semantic proximity Read ↗

Mutations in FAM134B, encoding a newly identified Golgi protein, cause severe sensory and autonomic neuropathy.Semantic proximity Read ↗

Zebrafish live imaging reveals a surprisingly small percentage of spinal cord motor neurons die during early developmentSemantic proximity Read ↗

EFF-1-mediated regenerative axonal fusion requires components of the apoptotic pathway.Semantic proximity Read ↗

In vivo assessment of neuronal cell death in Drosophila.Semantic proximity Read ↗

Developmental Attenuation of Neuronal Apoptosis by Neural-Specific Splicing of Bak1 Microexon.Semantic proximity Read ↗

A molecular pathway for light-dependent photoreceptor apoptosis in Drosophila.Semantic proximity Read ↗

Genetic analysis of soluble N-ethylmaleimide-sensitive factor attachment protein function in Drosophila reveals positive and negative secretory roles.Semantic proximity Read ↗

Sequential SNARE disassembly and GATE-16-GOS-28 complex assembly mediated by distinct NSF activities drives Golgi membrane fusion.Semantic proximity Read ↗

Cytotoxicity of botulinum neurotoxins reveals a direct role of syntaxin 1 and SNAP-25 in neuron survival.Semantic proximity Read ↗

Neuronal Apoptosis in Sudden Infant Death SyndromeSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.