Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The association between the catechol-O-methyltransferase Val108/158Met polymorphism and hyperactive-impulsive and inattentive symptoms in youth.

Psychopharmacology - 1 Nov 2013

Nikolac Perkovic Matea, Kiive Evelyn, Nedic Erjavec Gordana, Veidebaum Toomas, Curkovic Mario, Dodig-Curkovic Katarina, Muck-Seler Dorotea, Harro Jaanus, Pivac Nela

Abstract excerpt

RATIONALE: Hyperactivity, impulsivity, and inattention are major symptoms occurring in attention-deficit/hyperactivity disorder. This disorder is highly heritable, multifactorial, polygenic, and associated primarily with dysfunctions of dopaminergic, noradrenergic, and serotonergic systems. OBJECTIVES: The present study tested the possible association of the catechol-O-methyltransferase (COMT) Val108/158Met...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Attention Deficit Disorder with Hyperactivity
Share to Catechol O-Methyltransferase

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Dopamine

Share to Impulsive Behavior

Share to Psychometrics

Share to Severity of Illness Index

Share to Surveys and Questionnaires

Share to White People

Source

Identifiers

PMID: 23715639
DOI: 10.1007/s00213-013-3138-1

Genes

COMT

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The association between the catechol-O-methyltransferase Val108/158Met polymorphism and hyperactive-impulsive and inattentive symptoms in youth.PMID 23715639Read ↗

Attention-deficit/hyperactivity disorder phenotype is influenced by a functional catechol-O-methyltransferase variant.Semantic proximity Read ↗

Catechol-O-methyltransferase Val158-Met polymorphism and a response of hyperactive-impulsive symptoms to methylphenidate: A replication study from South Korea.Semantic proximity Read ↗

Association between catechol O-methyltransferase (COMT) haplotypes and severity of hyperactivity symptoms in adults.Semantic proximity Read ↗

Pathogenic study on catechol-O-methyltransferase gene and catecholaminergic neurotransmitters with attention deficit hyperactivity disorder in Chinese childrenSemantic proximity Read ↗

Association between the Catechol O-methyltransferase (COMT) Val158met polymorphism and different dimensions of impulsivity.Semantic proximity Read ↗

The met(158) allele of catechol-O-methyltransferase (COMT) is associated with obsessive-compulsive disorder in men: case-control study and meta-analysis.Semantic proximity Read ↗

Association of the catechol-O-methyltransferase val158met polymorphism and anxiety-related traits: a meta-analysis.Semantic proximity Read ↗

Val158Met polymorphisms of COMT gene and serum concentrations of catecholaminergic neurotransmitters of ADHD in Chinese children and adolescents.Semantic proximity Read ↗

Variation in the catechol-O-methyltransferase Val 158 Met polymorphism associated with conduct disorder and ADHD symptoms, among adolescent male delinquents.Semantic proximity Read ↗

Association of the COMT Met¹⁵⁸ allele with trait impulsivity in healthy young adults.Semantic proximity Read ↗

Association of Val158Met polymorphism in COMT gene with attention-deficit hyperactive disorder: An updated meta-analysis.Semantic proximity Read ↗

[Catechol-O-methyltransferase gene rs6267 polymorphism in children with attention deficit hyperactivity disorder].Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.