Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Expanded HIV-1 cellular tropism by phenotypic mixing with murine endogenous retroviruses.

Science (New York, N.Y.) - 16 Feb 1990

Lusso P, di Marzo Veronese F, Ensoli B, Franchini G, Jemma C, DeRocco S E, Kalyanaraman V S, Gallo R C

Abstract excerpt

In view of the current interest in in vivo murine models for acquired immunodeficiency syndrome (AIDS), the interaction between human immunodeficiency virus type 1 (HIV-1) and endogenous murine leukemia virus (MuLV)-related retroviruses was investigated with a human leukemic T cell line (PF-382x) that acquired xenotropic MuLV (X-MuLV) after in vivo passage in immunosuppressed mice. Despite similar levels of...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Antibodies, Monoclonal
Share to Cell Line

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Disease Models, Animal

Share to Phenotype

Share to Viral Proteins

Share to Virus Replication

Source

Identifiers

PMID: 2305256
DOI: 10.1126/science.2305256

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Expanded HIV-1 cellular tropism by phenotypic mixing with murine endogenous retroviruses.PMID 2305256Read ↗

Replication of HIV-1 in a wide variety of animal cells following phenotypic mixing with murine retroviruses.Semantic proximity Read ↗

CD4-independent infection by human immunodeficiency virus type 1 after phenotypic mixing with human T-cell leukemia viruses.Semantic proximity Read ↗

Coexistence of fusion receptors for human T-cell leukemia virus type-I (HTLV-I) and human immunodeficiency virus type-1 (HIV-1) on MOLT-4 cells.Semantic proximity Read ↗

Replicative fitness of CCR5-using and CXCR4-using human immunodeficiency virus type 1 biological clones.Semantic proximity Read ↗

Possible viral interactions in the acquired immunodeficiency syndrome (AIDS).Semantic proximity Read ↗

Fv-1 restriction of endogenous feline C-type RD114 virus genome phenotypically mixed with ecotropic murine leukemia viruses.Semantic proximity Read ↗

Human immunodeficiency virus (HIV) type 1 can superinfect HIV-2-infected cells: pseudotype virions produced with expanded cellular host range.Semantic proximity Read ↗

Distinct modes of human immunodeficiency virus type 1 proviral latency revealed by superinfection of nonproductively infected cell lines with recombinant luciferase-encoding viruses.Semantic proximity Read ↗

Human lymphoblastoid CD4(+) T cells become permissive to macrophage-tropic strains of human immunodeficiency virus type 1 after passage into severe combined immunodeficient mice through in vivo upregulation of CCR5: in vivo dynamics of CD4(+) T-cell differentiation in pathogenesis of AIDS.Semantic proximity Read ↗

The concurrent maturation of mouse and human thymocytes in human fetal thymus implanted in NIH-beige-nude-xid mice is associated with the reconstitution of the murine immune system.Semantic proximity Read ↗

Mouse retrovirus mediates porcine endogenous retrovirus transmission into human cells in long-term human-porcine chimeric miceSemantic proximity Read ↗

Fusogenic selectivity of the envelope glycoprotein is a major determinant of human immunodeficiency virus type 1 tropism for CD4+ T-cell lines vs. primary macrophages.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.