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Article

AKAP13, CACNA1, GRIK4 and GRIA1 genetic variations may be associated with haloperidol efficacy during acute treatment.

European neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology - 1 Aug 2013

Drago Antonio, Giegling Ina, Schäfer Martin, Hartmann Annette M, Friedl Marion, Konte Bettina, Möller Hans-Jürgen, De Ronchi Diana, Stassen Hans H, Serretti Alessandro, Rujescu Dan

Abstract excerpt

We previously investigated a sample of psychotic patients acutely ill and acutely treated with haloperidol in the search for genetic predictors of response at PANSS scores during the first month of treatment. In the present work we extend the analysis to a wider panel of genetic variations includ...

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Source

Identifiers

PMID: 22980146
DOI: 10.1016/j.euroneuro.2012.08.013

Genes

AKAP13
GRIA1
GRIK4

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026. Prepared record publication-22980146-2026-09-04.2. Manifest publication-graph-cohort-2026-09-04.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 4 Sept 2026.

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AKAP13, CACNA1, GRIK4 and GRIA1 genetic variations may be associated with haloperidol efficacy during acute treatment.PMID 22980146Read ↗

Glutamatergic gene variants impact the clinical profile of efficacy and side effects of haloperidol.Semantic proximity Read ↗

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Genome-wide association study supports the role of the immunological system and of the neurodevelopmental processes in response to haloperidol treatment.Semantic proximity Read ↗

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