Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Mechanisms of post-intervention arterial remodelling.

Cardiovascular research - 1 Dec 2012

Goel Shakti A, Guo Lian-Wang, Liu Bo, Kent K C

Abstract excerpt

It has been appreciated over the past two decades that arterial remodelling, in addition to intimal hyperplasia, contributes significantly to the degree of restenosis that develops following revascularization procedures. Remodelling appears to be an adventitia-based process that is contributed to by multiple factors including cytokines and growth factors that regulate extracellular matrix or phenotypic...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Arteries
Share to Coronary Artery Disease
Share to Coronary Restenosis

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Coronary Vessels

Share to Extracellular Matrix Proteins

Share to Neointima

Share to Percutaneous Coronary Intervention

Share to Peripheral Arterial Disease

Source

Open in PubMed Open full text

Identifiers

PMID: 22918976
DOI: 10.1093/cvr/cvs276
PMCID: PMC3500049

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Mechanisms of post-intervention arterial remodelling.PMID 22918976Read ↗

Effect of TGF-β1 on the Migration and Recruitment of Mesenchymal Stem Cells after Vascular Balloon Injury: Involvement of Matrix Metalloproteinase-14.Semantic proximity Read ↗

Biological responses in stented arteries.Semantic proximity Read ↗

Adventitial remodeling after coronary arterial injury.Semantic proximity Read ↗

The role of microRNAs in arterial remodelling.Semantic proximity Read ↗

CREG mediated adventitial fibroblast phenotype modulation: a possible therapeutic target for proliferative vascular disease.Semantic proximity Read ↗

The adventitia: a dynamic interface containing resident progenitor cells.Semantic proximity Read ↗

Cellular repressor E1A-stimulated genes controls phenotypic switching of adventitial fibroblasts by blocking p38MAPK activation.Semantic proximity Read ↗

Adventitial biology: differentiation and function.Semantic proximity Read ↗

Smooth muscle cell-specific Tgfbr1 deficiency attenuates neointimal hyperplasia but promotes an undesired vascular phenotype for injured arteries.Semantic proximity Read ↗

Phenotypic modulation of smooth muscle cells during formation of neointimal thickenings following vascular injury.Semantic proximity Read ↗

Smooth muscle cell signal transduction: implications of vascular biology for vascular surgeons.Semantic proximity Read ↗

Transforming growth factor-betas and vascular disorders.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.