Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Inflammatory Pathway Genes Belong to Major Targets of Persistent Organic Pollutants in Adipose Cells

19 Jan 2012

Abstract excerpt

BACKGROUND: Epidemiological studies emphasize the possible role of persistent organic pollutants (POPs) in obesity and the metabolic syndrome. These pollutants are stored in adipose tissue (AT). OBJECTIVES: Our aim was to study the effects of POPs on human adipose cells and rodent AT. METHODS: Using human multipotent adipose-derived stem cells, we carried out large-scale gene expression analysis to identify the...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adipokines, Inflammation, and Metabolic Diseases
Share to Effects and risks of endocrine disrupting chemicals
Share to Toxic Organic Pollutants Impact

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 22262711
DOI: 10.1289/ehp.1104282

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Inflammatory Pathway Genes Belong to Major Targets of Persistent Organic Pollutants in Adipose CellsPMID 22262711Read ↗

Human Preadipocytes Differentiated under Hypoxia following PCB126 Exposure during Proliferation: Effects on Differentiation, Glucose Uptake and Adipokine ProfileSemantic proximity Read ↗

Integrated transcriptomics and metabolomics reveal signatures of lipid metabolism dysregulation in HepaRG liver cells exposed to PCB 126Semantic proximity Read ↗

Differential gene expression and a functional analysis of PCB-exposed children: Understanding disease and disorder developmentSemantic proximity Read ↗

Toxicity impacts on human adipose MSCs acutely exposed to Aroclor and non-Aroclor mixtures of PCBsSemantic proximity Read ↗

Endocrine-Mediated Mechanisms of Metabolic Disruption and New Approaches to Examine the Public Health ThreatSemantic proximity Read ↗

Long-term in vivo polychlorinated biphenyl 126 exposure induces oxidative stress and alters proteomic profile on islets of LangerhansSemantic proximity Read ↗

Transcriptomic Analysis of Gonadal Adipose Tissue in Male Mice Exposed Perinatally to 2,2′,4,4′-Tetrabromodiphenyl Ether (BDE-47)Semantic proximity Read ↗

Cross-tissue molecular responses in the liver and blood after toxicant exposuresSemantic proximity Read ↗

ToxiTaRGET: a multi-omics resource for toxicant-responsive molecular targetsSemantic proximity Read ↗

Aryl hydrocarbon receptor SNP -130 C/T associates with dioxins susceptibility through regulating its receptor activity and downstream effectors including interleukin 24.Semantic proximity Read ↗

Transcriptional pathways linked to fetal and maternal hepatic dysfunction caused by gestational exposure to perfluorooctanoic acid (PFOA) or hexafluoropropylene oxide-dimer acid (HFPO-DA or GenX) in CD-1 miceSemantic proximity Read ↗

Air-pollutant chemicals and oxidized lipids exhibit genome-wide synergistic effects on endothelial cellsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.