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Article

RPS3a over-expressed in HBV-associated hepatocellular carcinoma enhances the HBx-induced NF-κB signaling via its novel chaperoning function.

PloS one - 1 Jan 2011

Lim Keo-Heun, Kim Kyun-Hwan, Choi Seong Il, Park Eun-Sook, Park Seung Hwa, Ryu Kisun, Park Yong Kwang, Kwon So Young, Yang Sung-Il, Lee Han Chu, Sung In-Kyung, Seong Baik L

Abstract excerpt

Hepatitis B virus (HBV) infection is one of the major causes of hepatocellular carcinoma (HCC) development. Hepatitis B virus X protein (HBx) is known to play a key role in the development of hepatocellular carcinoma (HCC). Several cellular proteins have been reported to be over-expressed in HBV-associated HCC tissues, but their role in the HBV-mediated oncogenesis remains largely unknown. Here, we explored the...

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Identifiers

PMID: 21857917
DOI: 10.1371/journal.pone.0022258
PMCID: PMC3156704

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

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