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Article

Expression and roles of Slit/Robo in human ovarian cancer

4 Apr 2011

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Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 21465248
DOI: 10.1007/s00418-011-0806-2

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Related research

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Expression and roles of Slit/Robo in human ovarian cancerPMID 21465248Read ↗

Slit2 signaling through Robo1 and Robo2 is required for retinal neovascularizationSemantic proximity Read ↗

Silencing of miRNA-218 promotes migration and invasion of breast cancer via Slit2-Robo1 pathwaySemantic proximity Read ↗

SLITs Suppress Tumor Growth <i>In vivo</i> by Silencing <i>Sdf1/Cxcr4</i> within Breast EpitheliumSemantic proximity Read ↗

Involvement of the SLIT/ROBO pathway in follicle development in the fetal ovarySemantic proximity Read ↗

Robo1/2 regulate follicle atresia through manipulating granulosa cell apoptosis in mice.Semantic proximity Read ↗

Robo signalling controls pancreatic progenitor identity by regulating Tead transcription factorsSemantic proximity Read ↗

Expression of the repulsive SLIT/ROBO pathway in the human endometrium and Fallopian tube.Semantic proximity Read ↗

SLIT2, a human homologue of the Drosophila Slit2 gene, has tumor suppressor activity and is frequently inactivated in lung and breast cancers.Semantic proximity Read ↗

Targeting the ROR1 and ROR2 receptors in epithelial ovarian cancer inhibits cell migration and invasionSemantic proximity Read ↗

Homeobox gene expression in ovarian cancer.Semantic proximity Read ↗

R-Spondin Proteins: A Novel Link to &beta;-catenin ActivationSemantic proximity Read ↗

Notch pathway promotes ovarian cancer growth and migration via CXCR4/SDF1α chemokine systemSemantic proximity Read ↗

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