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KCNQ1 gene variants and risk of new-onset diabetes in tacrolimus-treated renal-transplanted patients.

Clinical transplantation - 1 Jan 2000

Tavira Beatriz, Coto Eliecer, Díaz-Corte Carmen, Ortega Francisco, Arias Manuel, Torres Armando, Díaz Juan M, Selgas Rafael, López-Larrea Carlos, Campistol Jose M, Ruiz-Ortega Marta, Alvarez Victoria

Abstract excerpt

Recent genome-wide association studies identified single-nucleotide polymorphisms (SNPs) in the gene encoding the pore-forming subunit of the voltage-gated K+ channel (KCNQ1) as a risk factor for type 2 diabetes. Tacrolimus (Tac) increased the risk of new-onset diabetes after transplantation (NODAT). The aim of this study was to analyze the association between KCNQ1 variants and the risk for NODAT in...

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Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

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Source

Identifiers

PMID: 21355884
DOI: 10.1111/j.1399-0012.2011.01417.x

Genes

KCNQ1

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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