Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Inhibition of the c-fms proto-oncogene autocrine loop and tumor phenotype in glucocorticoid stimulated human breast carcinoma cells.

Breast cancer research and treatment - 1 Sept 2011

Toy Eugene P, Lamb Tiffany, Azodi Masoud, Roy William J, Woo Ho-Hyung, Chambers Setsuko K

Abstract excerpt

The c-fms proto-oncogene encoded CSF-1 receptor and its ligand represent a feedback loop, which in a paracrine manner, is well known to promote spread of breast cancers. The role of the autocrine feedback loop in promotion of breast tumor behavior, in particular in vitro, is less well understood....

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Autocrine Communication
Share to Dexamethasone
Share to Glucocorticoids

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Macrophage Colony-Stimulating Factor

Share to Oligonucleotides, Antisense

Share to Phenylurea Compounds

Share to Receptor, Macrophage Colony-Stimulating Factor

Share to Thiazoles

Source

Identifiers

PMID: 21063905
DOI: 10.1007/s10549-010-1247-7

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-21063905-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Inhibition of the c-fms proto-oncogene autocrine loop and tumor phenotype in glucocorticoid stimulated human breast carcinoma cells.PMID 21063905Read ↗

Correlation of tumor phenotype with c-fms proto-oncogene expression in an in vivo intraperitoneal model for experimental human breast cancer metastasis.Semantic proximity Read ↗

Hormonal regulation of the c-fms proto-oncogene in breast cancer cells is mediated by a composite glucocorticoid response element.Semantic proximity Read ↗

Independent regulation of invasion and anchorage-independent growth by different autophosphorylation sites of the macrophage colony-stimulating factor 1 receptor.Semantic proximity Read ↗

cDNA microarray analysis of invasive and tumorigenic phenotypes in a breast cancer model.Semantic proximity Read ↗

The impact of glucocorticoid receptor transactivation on context-dependent cell migration dynamicsSemantic proximity Read ↗

Effect of all-trans-retinoic acid on c-fms proto-oncogene [colony-stimulating factor 1 (CSF-1) receptor] expression and CSF-1-induced invasion and anchorage-independent growth of human breast carcinoma cells.Semantic proximity Read ↗

Differential signal transduction of alternatively spliced FGFR2 variants expressed in human mammary epithelial cells.Semantic proximity Read ↗

Shedding of syndecan-1 by stromal fibroblasts stimulates human breast cancer cell proliferation via FGF2 activation.Semantic proximity Read ↗

A chemical biology screen identifies glucocorticoids that regulate c-maf expression by increasing its proteasomal degradation through up-regulation of ubiquitinSemantic proximity Read ↗

Phenotypic conversion of human mammary carcinoma cells by autocrine human growth hormone.Semantic proximity Read ↗

Downregulation of SOK1 promotes the migration of MCF-7 cellsSemantic proximity Read ↗

Signal-response coupling mediated by the transduced colony-stimulating factor-1 receptor and its oncogenic fms variants in naive cells.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.