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Article

The psychiatric disease risk factors DISC1 and TNIK interact to regulate synapse composition and function

14 Sept 2010

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Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 20838393
DOI: 10.1038/mp.2010.87

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Related research

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Research trail→

Click a neighbor to travelStep 1 · 12 closest

The psychiatric disease risk factors DISC1 and TNIK interact to regulate synapse composition and functionPMID 20838393Read ↗

DISC1 at 10: connecting psychiatric genetics and neuroscience.Semantic proximity Read ↗

DISC1 genetics, biology and psychiatric illnessSemantic proximity Read ↗

Mitochondrial roles of the psychiatric disease risk factor DISC1.Semantic proximity Read ↗

The potential of DISC1 protein as a therapeutic target for mental illness.Semantic proximity Read ↗

Linking neurodevelopmental and synaptic theories of mental illness through DISC1.Semantic proximity Read ↗

DISCopathies: brain disorders related to DISC1 dysfunction.Semantic proximity Read ↗

Disrupted in Schizophrenia 1 (DISC1) is a multicompartmentalized protein that predominantly localizes to mitochondriaSemantic proximity Read ↗

Mechanisms underlying the role of DISC1 in synaptic plasticitySemantic proximity Read ↗

Regulation of mitochondrial dynamics by DISC1, a putative risk factor for major mental illness.Semantic proximity Read ↗

Proteomic Studies Reveal Disrupted in Schizophrenia 1 as a Key Regulator Unifying Neurodevelopment and Synaptic FunctionSemantic proximity Read ↗

DISC1 regulates synaptic vesicle transport via a lithium-sensitive pathwaySemantic proximity Read ↗

Disrupted-In-Schizophrenia-1 (DISC1) interactome and mental disorders: Impact of mouse modelsSemantic proximity Read ↗

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