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Article

Lack of association of the serotonin transporter polymorphism with the sudden infant death syndrome in the San Diego Dataset.

Pediatric research - 1 Nov 2010

Paterson David S, Rivera Keith D, Broadbelt Kevin G, Trachtenberg Felicia L, Belliveau Richard A, Holm Ingrid A, Haas Elisabeth A, Stanley Christina, Krous Henry F, Kinney Hannah C, Markianos Kyriacos

Abstract excerpt

Dysfunction of medullary serotonin (5-HT)-mediated respiratory and autonomic function is postulated to underlie the pathogenesis of the majority of sudden infant death syndrome (SIDS) cases. Several studies have reported an increased frequency of the LL genotype and L allele of the 5-HT transporter (5-HTT) gene promoter polymorphism (5-HTTLPR), which is associated with increased transcriptional activity and 5-HT...

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Identifiers

PMID: 20661167
DOI: 10.1203/PDR.0b013e3181f2edf0
PMCID: PMC3242414

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HTT

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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