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Article

Deficiency of Atf3, an adaptive-response gene, protects islets and ameliorates inflammation in a syngeneic mouse transplantation model

27 Mar 2010

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Share to Diabetes and associated disorders
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 20349223
DOI: 10.1007/s00125-010-1696-x

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Related research

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Click a neighbor to travelStep 1 · 12 closest

Deficiency of Atf3, an adaptive-response gene, protects islets and ameliorates inflammation in a syngeneic mouse transplantation modelPMID 20349223Read ↗

The Roles of ATF3 in Glucose HomeostasisSemantic proximity Read ↗

Master Regulator Activating Transcription Factor 3 (ATF3) in Metabolic Homeostasis and CancerSemantic proximity Read ↗

Role for Activating Transcription Factor 3 in Stress-Induced β-Cell ApoptosisSemantic proximity Read ↗

Activating transcription factor 3 regulates immune and metabolic homeostasis.Semantic proximity Read ↗

ATF3 expression in cardiomyocytes preserves homeostasis in the heart and controls peripheral glucose tolerance.Semantic proximity Read ↗

Systems biology approaches identify ATF3 as a negative regulator of Toll-like receptor 4Semantic proximity Read ↗

A20 is an immune tolerance factor that can determine islet transplant outcomesSemantic proximity Read ↗

Hypothalamic ATF3 is involved in regulating glucose and energy metabolism in mice.Semantic proximity Read ↗

Inducible ATF3–NFAT axis aggravates podocyte injurySemantic proximity Read ↗

The transcription factor ATF4 mediates endoplasmic reticulum stress-related podocyte injury and slit diaphragm defectsSemantic proximity Read ↗

Genetic Engineering of a Suboptimal Islet Graft with A20 Preserves β Cell Mass and FunctionSemantic proximity Read ↗

ATF4-Mediated Induction of 4E-BP1 Contributes to Pancreatic β Cell Survival under Endoplasmic Reticulum StressSemantic proximity Read ↗

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