Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Integrative genome-wide association analysis of cytoarchitectural abnormalities in the prefrontal cortex of psychiatric disorders.

Molecular psychiatry - 1 Apr 2011

Kim S, Webster M J

Abstract excerpt

Cytoarchitectural abnormalities have been described in the prefrontal cortex (PFC) of subjects with psychiatric disorders. We explored the possible genetic causalities that may underlie the cytoarchitectural abnormalities of calbindin-containing γ-aminobutyric acid (GABA)ergic neurons and perineuronal oligodendrocytes in the PFC of subjects with psychiatric disorders by converging results from genome-wide...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Calbindins
Share to Databases, Genetic
Share to gamma-Aminobutyric Acid

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Mental Disorders

Share to Oligodendroglia

Share to Prefrontal Cortex

Share to Protein Phosphatase 2

Share to S100 Calcium Binding Protein G

Source

Identifiers

PMID: 20308991
DOI: 10.1038/mp.2010.23

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Integrative genome-wide association analysis of cytoarchitectural abnormalities in the prefrontal cortex of psychiatric disorders.PMID 20308991Read ↗

Comprehensive analyses of RNA-seq and genome-wide data point to enrichment of neuronal cell type subsets in neuropsychiatric disordersSemantic proximity Read ↗

Connecting genomic results for psychiatric disorders to human brain cell types and regions reveals convergence with functional connectivitySemantic proximity Read ↗

Connecting genomic results for psychiatric disorders to human brain cell types and regions reveals convergence with functional connectivity.Semantic proximity Read ↗

Transcriptome-informed brain cartography of polygenic risk and association with brain structure in major psychiatric disorders.Semantic proximity Read ↗

Copy number variants reveal divergent genetic and diagnostic cortical signatures across psychiatric disordersSemantic proximity Read ↗

Combining NeuroPainting with transcriptomics reveals cell-type-specific morphological and molecular signatures of the 22q11.2 deletionSemantic proximity Read ↗

Decoding frontotemporal and cell type-specific vulnerabilities to neuropsychiatric disorders and psychoactive drugsSemantic proximity Read ↗

Gene set enrichment analysis and expression pattern exploration implicate an involvement of neurodevelopmental processes in bipolar disorder.Semantic proximity Read ↗

Quantitative cytoarchitectural phenotyping of deparaffinized human brain tissuesSemantic proximity Read ↗

Subtypes in 22q11.2 deletion syndrome associated with behaviour and neurofacial morphology.Semantic proximity Read ↗

Genetically distinct parallel projection populations from ventral hippocampus to prefrontal cortexSemantic proximity Read ↗

Combining phenomics with transcriptomics reveals cell-type-specific morphological and molecular signatures of the 22q11.2 deletionSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.