Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Transcriptome of normal lung distinguishes mouse lines with different susceptibility to inflammation and to lung tumorigenesis.

Cancer letters - 28 Aug 2010

Franco Marcelo De, Colombo Francesca, Galvan Antonella, Cecco Loris De, Spada Elena, Milani Silvano, Ibanez Olga M, Dragani Tommaso A

Abstract excerpt

AIRmax and AIRmin mouse lines show a differential lung inflammatory response and differential lung tumor susceptibility after urethane treatment. The transcript profile of approximately 24,000 known genes was analyzed in normal lung tissue of untreated and urethane-treated AIRmax and AIRmin mice. In lungs of untreated mice, inflammation-associated genes involved in pathways such as "leukocyte transendothelial...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cell Transformation, Neoplastic
Share to Disease Models, Animal

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Gene Expression

Share to Gene Expression Profiling

Share to Lung Neoplasms

Share to Pneumonia

Share to Urethane

Source

Identifiers

PMID: 20189714
DOI: 10.1016/j.canlet.2010.01.038

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Transcriptome of normal lung distinguishes mouse lines with different susceptibility to inflammation and to lung tumorigenesis.PMID 20189714Read ↗

Genetic selection for high acute inflammatory response confers resistance to lung carcinogenesis in the mouse.Semantic proximity Read ↗

Pulmonary adenoma susceptibility 1 (Pas1) locus affects inflammatory response.Semantic proximity Read ↗

Disease-Specific Gene Expression Profiling in Multiple Models of Lung DiseaseSemantic proximity Read ↗

Susceptibility to neoplastic and non-neoplastic pulmonary diseases in mice: genetic similarities.Semantic proximity Read ↗

The tobacco carcinogen nitrosamine induces a differential gene expression response in tumour susceptible A/J and resistant C3H mouse lungs.Semantic proximity Read ↗

Inflammation promotes aging-associated oncogenesis in the lungSemantic proximity Read ↗

Identification of a Common Gene Expression Response in Different Lung Inflammatory Diseases in Rodents and MacaquesSemantic proximity Read ↗

Identification of candidate lung cancer susceptibility genes in mouse using oligonucleotide arrays.Semantic proximity Read ↗

The Overlap of Lung Tissue Transcriptome of Smoke Exposed Mice with Human Smoking and COPDSemantic proximity Read ↗

Genetic heterogeneity of inflammatory response and skin tumorigenesis in phenotypically selected mouse lines.Semantic proximity Read ↗

Early gene expression differences in inbred mouse strains with susceptibility to pulmonary adenomas.Semantic proximity Read ↗

Effect of genetic modification of acute inflammatory responsiveness on tumorigenesis in the mouseSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.