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Article

Myoglobin-deficient mice activate a distinct cardiac gene expression program in response to isoproterenol-induced hypertrophy.

Physiological genomics - 1 Apr 2010

Molojavyi Andrei, Lindecke Antje, Raupach Annika, Moellendorf Sarah, Köhrer Karl, Gödecke Axel

Abstract excerpt

Myoglobin knockout mice (myo-/-) adapt to the loss of myoglobin by the activation of a variety of compensatory mechanisms acting on the structural and functional level. To analyze to what extent myo-/- mice would tolerate cardiac stress we used the model of chronic isoproterenol application to in...

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Source

Identifiers

PMID: 20145201
DOI: 10.1152/physiolgenomics.90297.2008

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-20145201-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

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