Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Childhood brain tumors, residential insecticide exposure, and pesticide metabolism genes.

Environmental health perspectives - 1 Jan 2010

Searles Nielsen Susan, McKean-Cowdin Roberta, Farin Federico M, Holly Elizabeth A, Preston-Martin Susan, Mueller Beth A

Abstract excerpt

BACKGROUND: Insecticides that target the nervous system may play a role in the development of childhood brain tumors (CBTs). Constitutive genetic variation affects metabolism of these chemicals. METHODS: We analyzed population-based case-control data to examine whether CBT is associated with the...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Aldehyde Dehydrogenase
Share to Aryldialkylphosphatase
Share to Butyrylcholinesterase

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Environmental Exposure

Share to Inactivation, Metabolic

Share to Insecticides

Share to Oxygenases

Share to Pesticides

Source

Open in PubMed Open full text

Identifiers

PMID: 20056567
DOI: 10.1289/ehp.0901226
PMCID: PMC2831959

Genes

FMO1
PON1
GSTT1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-20056567-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Childhood brain tumors, residential insecticide exposure, and pesticide metabolism genes.PMID 20056567Read ↗

Risk of brain tumors in children and susceptibility to organophosphorus insecticides: the potential role of paraoxonase (PON1).Semantic proximity Read ↗

Critical confluence: gene variants, insecticide exposure may increase childhood brain tumor risk.Semantic proximity Read ↗

MDR1 gene variants, indoor insecticide exposure, and the risk of childhood acute lymphoblastic leukemia.Semantic proximity Read ↗

Risk of childhood leukemia associated with exposure to pesticides and with gene polymorphisms.Semantic proximity Read ↗

Association between pesticide exposure and paraoxonase-1 (PON1) polymorphisms, and neurobehavioural outcomes in children: a systematic review.Semantic proximity Read ↗

What causes childhood brain tumors? Limited knowledge, many clues.Semantic proximity Read ↗

Variation in xenobiotic transport and metabolism genes, household chemical exposures, and risk of childhood acute lymphoblastic leukemia.Semantic proximity Read ↗

Polymorphisms of pesticide-metabolizing genes in children living in intensive farming communities.Semantic proximity Read ↗

Paraoxonase 1, agricultural organophosphate exposure, and Parkinson disease.Semantic proximity Read ↗

An exploratory case-only analysis of gene-hazardous air pollutant interactions and the risk of childhood medulloblastoma.Semantic proximity Read ↗

Repeated gestational exposure of mice to chlorpyrifos oxon is associated with paraoxonase 1 (PON1) modulated effects in maternal and fetal tissues.Semantic proximity Read ↗

Paraoxonase gene variants are associated with autism in North America, but not in Italy: possible regional specificity in gene-environment interactions.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.