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Article

KIF26A Is an Unconventional Kinesin and Regulates GDNF-Ret Signaling in Enteric Neuronal Development

1 Nov 2009

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Share to Congenital gastrointestinal and neural anomalies
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 19914172
DOI: 10.1016/j.cell.2009.10.023

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Related research

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KIF26A Is an Unconventional Kinesin and Regulates GDNF-Ret Signaling in Enteric Neuronal DevelopmentPMID 19914172Read ↗

KIF26A is mutated in the syndrome of congenital hydrocephalus with megacolon.Semantic proximity Read ↗

KIF1A/UNC-104 Transports ATG-9 to Regulate Neurodevelopment and Autophagy at SynapsesSemantic proximity Read ↗

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<i>De novo</i> mutations in KIF1A-associated neuronal disorder (KAND) dominant-negatively inhibit motor activity and axonal transport of synaptic vesicle precursorsSemantic proximity Read ↗

A systems-level proteomic analysis identifies kinesin targets of KIFBP during neuronal developmentSemantic proximity Read ↗

Kinesins in Caenorhabditis elegans neuronal morphogenesis.Semantic proximity Read ↗

De novo mutations in KIF1A-associated neuronal disorder (KAND) dominant-negatively inhibit motor activity and axonal transport of synaptic vesicle precursors.Semantic proximity Read ↗

Transcription and Signaling Regulators in Developing Neuronal Subtypes of Mouse and Human Enteric Nervous System.Semantic proximity Read ↗

Kinesin-1 acts with netrin and DCC to maintain sensory neuron position in Caenorhabditis elegans.Semantic proximity Read ↗

One gene, many phenotypes: the role of KIF5A in neurodegenerative and neurodevelopmental diseases.Semantic proximity Read ↗

CRMP/UNC-33 organizes microtubule bundles for KIF5-mediated mitochondrial distribution to axon.Semantic proximity Read ↗

TALPID3/KIAA0586 regulates multiple aspects of neuromuscular patterning during gastrointestinal development in animal models and humanSemantic proximity Read ↗

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