Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Epstein-Barr virus BZLF1 gene promoter variants in pediatric patients with acute infectious mononucleosis: its comparison with pediatric lymphomas.

Journal of medical virology - 1 Nov 2009

Lorenzetti Mario Alejandro, Gutiérrez Marina Inés, Altcheh Jaime, Moscatelli Guillermo, Moroni Samanta, Chabay Paola Andrea, Preciado María Victoria

Abstract excerpt

Epstein-Barr virus genotypes can be distinguished by polymorphic variations in the genes encoding EBNA2, 3A, 3B, and 3C. The immediate early gene BZLF1 plays a key role in modulating the switch from latency to lytic replication and therefore enabling viral propagation. The aim of this study was to investigate and compare BZLF1 promoter sequence (Zp) variation in pediatric infectious mononucleosis (IM) and in...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Biopsy
Share to Child, Preschool

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Herpesvirus 4, Human

Share to Leukocytes, Mononuclear

Share to Lymphoma

Share to Pharynx

Share to Promoter Regions, Genetic

Share to Trans-Activators

Source

Identifiers

PMID: 19774688
DOI: 10.1002/jmv.21616

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Epstein-Barr virus BZLF1 gene promoter variants in pediatric patients with acute infectious mononucleosis: its comparison with pediatric lymphomas.PMID 19774688Read ↗

LMP1 promoter sequence analysis in Epstein Barr virus pediatric infection reveals preferential circulation of B95.8 related variants in Argentina.Semantic proximity Read ↗

EBNA1 sequences in Argentinean pediatric acute and latent Epstein-Barr virus infection reflect circulation of novel South American variants.Semantic proximity Read ↗

[Characterization of BZLF1 gene and its promoter Zp of EBV strains in children with EBV-associated diseases in recent 5 years in Beijing area].Semantic proximity Read ↗

Discrete Alterations in the BZLF1 Promoter in Tumor and Non-Tumor-Associated Epstein-Barr VirusSemantic proximity Read ↗

A cancer-associated Epstein-Barr virus BZLF1 promoter variant enhances lytic infection.Semantic proximity Read ↗

Characterization of Epstein-Barr virus (EBV) BZLF1 gene promoter variants and comparison of cellular gene expression profiles in Japanese patients with infectious mononucleosis, chronic active EBV infection, and EBV-associated hemophagocytic lymphohistiocytosis.Semantic proximity Read ↗

Characterization of variants in the promoter of BZLF1 gene of EBV in nonmalignant EBV-associated diseases in Chinese childrenSemantic proximity Read ↗

Evidence of latent-lytic replication in EBV-positive Burkitt lymphoma from whole genome and transcriptome sequencingSemantic proximity Read ↗

Epstein-Barr virus from Burkitt Lymphoma biopsies from Africa and South America share novel LMP-1 promoter and gene variations.Semantic proximity Read ↗

Epstein Barr virus genomes reveal population structure and type 1 association with endemic Burkitt lymphomaSemantic proximity Read ↗

Reactivation of Epstein-Barr virus: regulation and function of the BZLF1 geneSemantic proximity Read ↗

Distribution of Epstein–Barr Virus LMP1 Variants in Patients with Infectious Mononucleosis and Association with Selected Biochemical and Hematological ParametersSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.