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Article

Modulation of glucocorticoid receptor nuclear translocation in neurons by immunophilins FKBP51 and FKBP52: Implications for major depressive disorder

22 Jun 2009

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Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Identifiers

PMID: 19545546
DOI: 10.1016/j.brainres.2009.06.036

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Modulation of glucocorticoid receptor nuclear translocation in neurons by immunophilins FKBP51 and FKBP52: Implications for major depressive disorderPMID 19545546Read ↗

Regulation of the glucocorticoid response to stress‐related disorders by the Hsp90‐binding immunophilin FKBP51Semantic proximity Read ↗

Differential Dynamics and Roles of FKBP51 Isoforms and Their Implications for Targeted TherapiesSemantic proximity Read ↗

Role of FKBP5 and its genetic mutations in stress-induced psychiatric disorders: an opportunity for drug discoverySemantic proximity Read ↗

FKBP51—a selective modulator of glucocorticoid and androgen sensitivitySemantic proximity Read ↗

The role of FKBP5, a co-chaperone of the glucocorticoid receptor in the pathogenesis and therapy of affective and anxiety disorders.Semantic proximity Read ↗

Targeting glucocorticoid receptor signaling pathway for treatment of stress-related brain disordersSemantic proximity Read ↗

Gene-Stress-Epigenetic Regulation of FKBP5: Clinical and Translational Implications.Semantic proximity Read ↗

The Role of FKBP5 in Mood Disorders: Action of FKBP5 on Steroid Hormone Receptors Leads to Questions About its Evolutionary ImportanceSemantic proximity Read ↗

Targeted ablation reveals a novel role of FKBP52 in gene-specific regulation of glucocorticoid receptor transcriptional activitySemantic proximity Read ↗

The prospect of FKBP51 as a drug target.Semantic proximity Read ↗

Differential Dynamics and Roles of FKBP51 Isoforms and Their Implications for Targeted TherapiesSemantic proximity Read ↗

Hsp90 Heterocomplexes Regulate Steroid Hormone Receptors: From Stress Response to Psychiatric DiseaseSemantic proximity Read ↗

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