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Article

BCR-ABL truncation due to premature translation termination as a mechanism of resistance to kinase inhibitors.

Acta haematologica - 1 Jan 2009

Ma Wanlong, Kantarjian Hagop, Yeh Chen-Hsiung, Zhang Zhong J, Cortes Jorge, Albitar Maher

Abstract excerpt

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Topics

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Share to Drug Resistance, Neoplasm
Share to Fusion Proteins, bcr-abl
Share to BCR gene
Share to Leukemia, Myelogenous, Chronic, BCR-ABL Positive

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Mutation

Share to Peptide Chain Termination, Translational

Share to Philadelphia Chromosome

Share to Protein Kinase Inhibitors

Source

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Identifiers

PMID: 19332983
DOI: 10.1159/000210060
PMCID: PMC2790522

Genes

BCR

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

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BCR-ABL truncation due to premature translation termination as a mechanism of resistance to kinase inhibitors.PMID 19332983Read ↗

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