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Article

Infection by Helicobacter pylori expressing the BabA adhesin is influenced by the secretor phenotype.

The Journal of pathology - 1 Jul 2008

Azevedo M, Eriksson S, Mendes N, Serpa J, Figueiredo C, Resende L P, Ruvoën-Clouet N, Haas R, Borén T, Le Pendu J, David L

Abstract excerpt

Helicobacter pylori (Hp) infects half the world's population and causes diverse gastric lesions, from gastritis to gastric cancer. Our aim was to evaluate the significance of secretor and Lewis status in infection and in vitro adherence by Hp expressing BabA adhesin. We enrolled 304 Hp-infected individuals from Northern Portugal. Gastric biopsies, blood and saliva were collected. Polymerase chain reaction (PCR)...

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Share to Adhesins, Bacterial
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Fucosyltransferases

Share to Galactoside 2-alpha-L-fucosyltransferase

Share to Gastric Mucosa

Share to Lewis Blood Group Antigens

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Source

Identifiers

PMID: 18498114
DOI: 10.1002/path.2363

Genes

FUT2

Variants

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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