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Article

Estrogen stimulates transcription from the human prolactin distal promoter through AP1 and estrogen responsive elements in T47D human breast cancer cells

20 Nov 2007

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Identifiers

PMID: 18022314
DOI: 10.1016/j.mce.2007.10.004

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Estrogen stimulates transcription from the human prolactin distal promoter through AP1 and estrogen responsive elements in T47D human breast cancer cellsPMID 18022314Read ↗

Human Prolactin Gene Promoter Regulation by Estrogen: Convergence with Tumor Necrosis Factor-α SignalingSemantic proximity Read ↗

Prolactin receptor gene transcriptional control, regulatory modalities relevant to breast cancer resistance and invasivenessSemantic proximity Read ↗

Cloning and Characterization of Human Estrogen Receptor β PromoterSemantic proximity Read ↗

Nonpituitary human prolactin gene transcription is independent of Pit-1 and differentially controlled in lymphocytes and in endometrial stromaSemantic proximity Read ↗

Complex 5′ Genomic Structure of the Human Prolactin Receptor: Multiple Alternative Exons 1 and Promoter UtilizationSemantic proximity Read ↗

Characterization of an up-stream promoter directing extrapituitary expression of the human prolactin geneSemantic proximity Read ↗

Prolactin signals via Stat5 and Oct-1 to the proximal cyclin D1 promoter.Semantic proximity Read ↗

Characterization of an up-stream promoter directing extrapituitary expression of the human prolactin gene.Semantic proximity Read ↗

Role of Estrogen Response Element in the Human Prolactin Gene: Transcriptional Response and Timing.Semantic proximity Read ↗

Identification of a Hormone-Responsive Promoter Immediately Upstream of Exon 1c in the Human Vitamin D Receptor Gene*Semantic proximity Read ↗

Regulation of estrogen receptor gene mediated by promoter B responsible for its enhanced expression in human breast cancerSemantic proximity Read ↗

Two distinct estrogen‐regulated promoters generate transcripts encoding the two functionally different human progesterone receptor forms A and B.Semantic proximity Read ↗

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