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Article

Complete deletion of all alpha-dystrobrevin isoforms does not reveal new neuromuscular junction phenotype.

Gene expression - 1 Jan 2007

Wang Dongqing, Kelly Bridget B, Albrecht Douglas E, Adams Marvin E, Froehner Stanley C, Feng Guoping

Abstract excerpt

The dystrophin glycoprotein complex (DGC) is critical for muscle stability, and mutations in DGC proteins lead to muscular dystrophy. The DGC also contributes to the maturation and maintenance of the neuromuscular junction (NMJ). The gene encoding the DGC protein alpha-dystrobrevin undergoes alternative splicing to produce at least five known isoforms. Isoform-specific antibody staining and reverse transcription...

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Identifiers

PMID: 17933218
DOI: 10.3727/000000007783991745
PMCID: PMC6042018

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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