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Article

FGF9 regulates early hypertrophic chondrocyte differentiation and skeletal vascularization in the developing stylopod.

Developmental biology - 15 Jul 2007

Hung Irene H, Yu Kai, Lavine Kory J, Ornitz David M

Abstract excerpt

Gain-of-function mutations in fibroblast growth factor (FGF) receptors result in chondrodysplasia and craniosynostosis syndromes, highlighting the critical role for FGF signaling in skeletal development. Although the FGFRs involved in skeletal development have been well characterized, only a single FGF ligand, FGF18, has been identified that regulates skeletal development during embryogenesis. Here we identify...

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Topics

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Share to Bone Development
Share to Chondrocytes
Share to Chondrogenesis

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Embryo Culture Techniques

Share to Fibroblast Growth Factors

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Share to Parathyroid Hormone-Related Protein

Share to Receptor, Fibroblast Growth Factor, Type 3

Source

Open in PubMed Open full text

Identifiers

PMID: 17544391
DOI: 10.1016/j.ydbio.2007.04.048
PMCID: PMC2267922

Genes

FGF9
FGF18
FGFR3

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026.

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