Dense SNP association study for bipolar I disorder on chromosome 18p11 suggests two loci with excess paternal transmission.

doi:10.1038/sj.mp.4001916

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Article

Dense SNP association study for bipolar I disorder on chromosome 18p11 suggests two loci with excess paternal transmission.

Molecular psychiatry - 1 Apr 2007

Mulle J G, Fallin M D, Lasseter V K, McGrath J A, Wolyniec P S, Pulver A E

Abstract excerpt

Parent-of-origin effects have been implicated as mediators of genetic susceptibility for a number of complex disease phenotypes, including bipolar disorder. Specifically, evidence for linkage on chromosome 18 is modified when allelic parent-of-origin is accommodated in the analysis. Our goal was to characterize the susceptibility locus for bipolar I disorder on chromosome 18p11 and investigate this...

Topics

Alleles
Bipolar Disorder
Chromosomes, Human, Pair 18
Family Health
Father-Child Relations
Female
Gene Frequency
Genetic Predisposition to Disease
Genotype
Humans
Jews
Linkage Disequilibrium
Longitudinal Studies
Male
Polymorphism, Single Nucleotide

Source

Open in PubMed

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PMID: 17389904
DOI: 10.1038/sj.mp.4001916

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