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Article

Loss of the tumor suppressor Vhlh leads to upregulation of Cxcr4 and rapidly progressive glomerulonephritis in mice

13 Aug 2006

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Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Identifiers

PMID: 16906157
DOI: 10.1038/nm1460

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

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Loss of the tumor suppressor Vhlh leads to upregulation of Cxcr4 and rapidly progressive glomerulonephritis in micePMID 16906157Read ↗

VHL loss reprograms the immune landscape to promote an inflammatory myeloid microenvironment in renal tumorigenesisSemantic proximity Read ↗

Conditional inactivation of the mouse von Hippel-Lindau tumor suppressor gene results in wide-spread hyperplastic, inflammatory and fibrotic lesions in the kidney.Semantic proximity Read ↗

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The von Hippel-Lindau Tumor Suppressor Stabilizes Novel Plant Homeodomain Protein Jade-1Semantic proximity Read ↗

Vhl deletion in renal epithelia causes HIF-1α-dependent, HIF-2α-independent angiogenesis and constitutive diuresis.Semantic proximity Read ↗

Combined VHLH and PTEN mutation causes genital tract cystadenoma and squamous metaplasia.Semantic proximity Read ↗

Regulation of CXCR4 signalingSemantic proximity Read ↗

STAT4 deficiency reduces autoantibody production and glomerulonephritis in a mouse model of lupusSemantic proximity Read ↗

Drosophila von Hippel-Lindau tumor suppressor gene function in epithelial tubule morphogenesis.Semantic proximity Read ↗

Regulation of renal epithelial tight junctions by the von Hippel-Lindau tumor suppressor gene involves occludin and claudin 1 and is independent of E-cadherin.Semantic proximity Read ↗

Tumor suppressor von Hippel-Lindau (VHL) stabilization of Jade-1 protein occurs through plant homeodomains and is VHL mutation dependent.Semantic proximity Read ↗

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