Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Consistent effects of TSG101 genetic variability on multiple outcomes of exposure to human immunodeficiency virus type 1.

Journal of virology - 1 Jul 2006

Bashirova Arman A, Bleiber Gabriela, Qi Ying, Hutcheson Holli, Yamashita Traci, Johnson Randall C, Cheng Jie, Alter Galit, Goedert James J, Buchbinder Susan, Hoots Keith, Vlahov David, May Margaret, Maldarelli Frank, Jacobson Lisa, O'brien Stephen J, Telenti Amalio, Carrington Mary

Abstract excerpt

Tumor susceptibility gene 101 (TSG101) encodes a host cellular protein that is appropriated by human immunodeficiency virus type 1 (HIV-1) in the budding process of viral particles from infected cells. Variation in the coding or noncoding regions of the gene could potentially affect the degree of TSG101-mediated release of viral particles. While the coding regions of the gene were found to lack nonsynonymous...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Acquired Immunodeficiency Syndrome
Share to CD4-Positive T-Lymphocytes

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Disease Progression

Share to Endosomal Sorting Complexes Required for Transport

Share to Open Reading Frames

Share to Viral Load

Share to Virus Shedding

Source

Open in PubMed Open full text

Identifiers

PMID: 16809281
DOI: 10.1128/jvi.00094-06
PMCID: PMC1489017

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Consistent effects of TSG101 genetic variability on multiple outcomes of exposure to human immunodeficiency virus type 1.PMID 16809281Read ↗

CD101 genetic variants modify regulatory and conventional T cell phenotypes and functions.Semantic proximity Read ↗

Variability in the P6gag domains of HIV-1 involved in viral budding.Semantic proximity Read ↗

Whole genome sequencing of extreme phenotypes identifies variants in CD101 and UBE2V1 associated with increased risk of sexually acquired HIV-1.Semantic proximity Read ↗

Association of BST-2 gene variants with HIV disease progression underscores the role of BST-2 in HIV type 1 infection.Semantic proximity Read ↗

The impact of human genetic variation on HIV disease in the era of HAART.Semantic proximity Read ↗

Genomewide association study of a rapid progression cohort identifies new susceptibility alleles for AIDS (ANRS Genomewide Association Study 03).Semantic proximity Read ↗

Risk association of BST2 gene variants with disease progression in HIV-1 infected Indian cohort.Semantic proximity Read ↗

Mapping variants in HTLV-1 genome to analyze their impacts on the HAM/TSP development: A systematic review.Semantic proximity Read ↗

Fraction of cases of acquired immunodeficiency syndrome prevented by the interactions of identified restriction gene variants.Semantic proximity Read ↗

Association between regulated upon activation, normal T cells expressed and secreted (RANTES) -28C/G polymorphism and susceptibility to HIV-1 infection: a meta-analysis.Semantic proximity Read ↗

Development of co-selected single nucleotide polymorphisms in the viral promoter precedes the onset of human immunodeficiency virus type 1-associated neurocognitive impairment.Semantic proximity Read ↗

Genetic variants in the host restriction factor APOBEC3G are associated with HIV-1-related disease progression and central nervous system impairment in children.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.