Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The animal pathogen-like type III secretion system is required for the intracellular survival of Burkholderia mallei within J774.2 macrophages.

Infection and immunity - 1 Jul 2006

Ribot Wilson J, Ulrich Ricky L

Abstract excerpt

Burkholderia mallei is a highly infectious gram-negative pathogen and is the causative agent of human and animal glanders. By generating polar mutations (disruption of bsaQ and bsaZ) in the B. mallei ATCC 23344 animal pathogen-like type III secretion system (TTS), we demonstrate that this bacteri...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cell Line
Share to Intracellular Fluid
Share to Lysosomal-Associated Membrane Protein 1

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Macrophages

Share to Mutagenesis

Share to Mutation

Source

Open in PubMed Open full text

Identifiers

PMID: 16790809
DOI: 10.1128/IAI.01939-05
PMCID: PMC1489733

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-16790809-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The animal pathogen-like type III secretion system is required for the intracellular survival of Burkholderia mallei within J774.2 macrophages.PMID 16790809Read ↗

Burkholderia mallei cluster 1 type VI secretion mutants exhibit growth and actin polymerization defects in RAW 264.7 murine macrophages.Semantic proximity Read ↗

Autotransporters and Their Role in the Virulence of Burkholderia pseudomallei and Burkholderia malleiSemantic proximity Read ↗

An Inv/Mxi‐Spa‐like type III protein secretion system in <i>Burkholderia pseudomallei</i> modulates intracellular behaviour of the pathogenSemantic proximity Read ↗

Type III Secretion: a Virulence Factor Delivery System Essential for the Pathogenicity of Burkholderia malleiSemantic proximity Read ↗

Novel Burkholderia mallei Virulence Factors Linked to Specific Host-Pathogen Protein InteractionsSemantic proximity Read ↗

Strategies for Intracellular Survival of Burkholderia pseudomalleiSemantic proximity Read ↗

<i>Burkholderia pseudomallei</i> BicA protein promotes pathogenicity in macrophages by regulating Invasion, Intracellular Survival, and VirulenceSemantic proximity Read ↗

Molecular Insights into <i>Burkholderia pseudomallei</i> and <i>Burkholderia mallei</i> PathogenesisSemantic proximity Read ↗

Outer Membrane Proteome of <i>Burkholderia pseudomallei</i> and <i>Burkholderia mallei</i> From Diverse Growth ConditionsSemantic proximity Read ↗

Inactivation of Burkholderia pseudomallei bsaQ results in decreased invasion efficiency and delayed escape of bacteria from endocytic vesicles.Semantic proximity Read ↗

Pathogenicity and virulence ofBurkholderia pseudomalleiSemantic proximity Read ↗

Comparative assessment of the intracellular survival of the Burkholderia pseudomallei bopC mutant.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.