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Article

An enzymatic ruler modulates Lewis antigen glycosylation of <i>Helicobacter pylori</i> LPS during persistent infection

13 Feb 2006

Abstract excerpt

Helicobacter pylori persistently colonizes about half the human population and contributes to the development of peptic ulcer disease and gastric cancer. This organism has evolved means to structurally alter its surface characteristics to evade innate and adaptive immune responses. H. pylori produces LPS O-antigen units that can be posttranslationally fucosylated to generate Lewis antigens, structures also found...

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PMID: 16477004
DOI: 10.1073/pnas.0511119103

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