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Article

Mouse Dach2 mutants do not exhibit gross defects in eye development or brain function.

Genesis (New York, N.Y. : 2000) - 1 Feb 2006

Davis Richard J, Pesah Yakov I, Harding Mark, Paylor Richard, Mardon Graeme

Abstract excerpt

Drosophila dachshund is a critical regulator of eye, brain, and limb formation. Vertebrate homologs, Dach1 and Dach2, are expressed in the developing retina, brain, and limbs, suggesting functional conservation of the dachshund/Dach gene family. Dach1 mutants die postnatally, but exhibit grossly normal development. Here we report the generation of Dach2 mutant mice. Although deletion of Dach2 exon 1 results in...

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Identifiers

PMID: 16470613
DOI: 10.1002/gene.20188

Genes

DACH2

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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