Article
Molecular genetics of unconjugated hyperbilirubinemia in Taiwanese.
Journal of biomedical science - 1 Jan 2005
Huang Ching-Shan
Abstract excerpt
In bilirubin metabolism, increased destruction of erythrocytes, defect in the function of organic anion transporter polypeptide 2 (OATP2) or UDP-glucuronosyltransferase 1A1 (UGT1A1) may result in unconjugated hyperbilirubinemia. Although glucose-6-phosphate dehydrogenase (G6PD) deficiency is known to be associated with the development of neonatal hyperbilirubinemia, it was observed that in neonates severe...
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