Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Plasma levels of tissue factor and soluble E-selectin in sickle cell disease: relationship to genotype and to inflammation.

Blood coagulation & fibrinolysis : an international journal in haemostasis and thrombosis - 1 Apr 2005

Mohan Junette S, Lip Gregory Yh, Wright Josh, Bareford David, Blann Andrew D

Abstract excerpt

BACKGROUND: Microvascular occlusion, the pathophysiological hallmark of sickle cell disease (SCD), is a complex multifactorial process with alterations in coagulation, endothelial function and inflammation. However, relationships between these process in the two most common genotypes, HbSS and HbSC, are unknown. We hypothesized differences in the hypercoagulable state [as assessed by tissue factor (TF),...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Anemia, Sickle Cell
Share to C-Reactive Protein

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to E-Selectin

Share to Fibrin Fibrinogen Degradation Products

Share to Inflammation

Share to Interleukin-6

Share to Thromboplastin

Share to von Willebrand Factor

Source

Identifiers

PMID: 15795541
DOI: 10.1097/01.mbc.0000164431.98169.8f

Genes

SCD

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Plasma levels of tissue factor and soluble E-selectin in sickle cell disease: relationship to genotype and to inflammation.PMID 15795541Read ↗

Soluble P-selectin and vascular endothelial growth factor in steady state sickle cell disease: relationship to genotype.Semantic proximity Read ↗

Endothelial Nitric Oxide Synthase (eNOS) Gene Polymorphisms and Markers of Hemolysis, Inflammation and Endothelial Dysfunction in Brazilian Sickle Cell Anemia Patients.Semantic proximity Read ↗

Platelet P-selectin and platelet mass, volume and component in sickle cell disease: relationship to genotype.Semantic proximity Read ↗

Influence of Hemoglobin Variants on Vaso-Occlusive Phenomena in Sickle Cell Anemia: A ReviewSemantic proximity Read ↗

Adhesion molecules and high-sensitivity C-reactive protein levels in patients with sickle cell beta-thalassaemia.Semantic proximity Read ↗

Markers of endothelial dysfunction differ between subphenotypes in children with sickle cell disease.Semantic proximity Read ↗

The role of blood rheology in sickle cell disease.Semantic proximity Read ↗

The endothelial biology of sickle cell disease: inflammation and a chronic vasculopathy.Semantic proximity Read ↗

l-selectin gene polymorphisms and complications of sickle cell disease.Semantic proximity Read ↗

Newer Aspects of the Pathophysiology of Sickle Cell Disease Vaso-OcclusionSemantic proximity Read ↗

Association of cell adhesion molecules levels & single nucleotide polymorphisms with vaso-occlusive crisis in sickle cell disease: A cross sectional study.Semantic proximity Read ↗

The clinical impact of MTHFR polymorphism on the vascular complications of sickle cell disease.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.