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Article

STAT5 regulates the self-renewal capacity and differentiation of human memory B cells and controls Bcl-6 expression

13 Feb 2005

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Share to Cytokine Signaling Pathways and Interactions
Share to Immune Cell Function and Interaction
Share to T-cell and B-cell Immunology

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 15711548
DOI: 10.1038/ni1172

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Related research

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STAT5 regulates the self-renewal capacity and differentiation of human memory B cells and controls Bcl-6 expressionPMID 15711548Read ↗

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STAT5B: A Differential Regulator of the Life and Death of CD4+ Effector Memory T CellsSemantic proximity Read ↗

STAT5 represses BCL6 expression by binding to a regulatory region frequently mutated in lymphomas.Semantic proximity Read ↗

STAT5b: A master regulator of key biological pathwaysSemantic proximity Read ↗

B Cell Lymphoma 6 (BCL6): A Conserved Regulator of Immunity and BeyondSemantic proximity Read ↗

STAT3 Role in T-Cell Memory FormationSemantic proximity Read ↗

Fc Receptor–like 5 Expression Distinguishes Two Distinct Subsets of Human Circulating Tissue–like Memory B CellsSemantic proximity Read ↗

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The T-ALL related gene BCL11B regulates the initial stages of human T-cell differentiationSemantic proximity Read ↗

Stat5-deficient hematopoiesis is permissive for Myc-induced B-cell leukemogenesis.Semantic proximity Read ↗

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