Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Cellular bases of activity-dependent paralysis in Drosophila stress-sensitive mutants.

Journal of neurobiology - 5 Sept 2004

Trotta Nick, Rodesch Chris K, Fergestad Tim, Broadie Kendal

Abstract excerpt

Stress-sensitive mutants in Drosophila have been shown to exhibit activity-dependent defects in neurotransmission. Using the neuromuscular junction (NMJ), this study investigates synaptic function more specifically in two stress-sensitive mutants: stress-sensitive B (sesB), which encodes a mitochondrial ADP/ATP translocase (ANT); and Atpalpha(2206), a conditional mutant of the Na+/K+ ATPase alpha-subunit....

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Evoked Potentials
Share to Excitatory Postsynaptic Potentials
Share to Paralysis

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Physical Stimulation

Share to Presynaptic Terminals

Share to Pyridinium Compounds

Share to Quaternary Ammonium Compounds

Share to Synaptic Vesicles

Source

Identifiers

PMID: 15281071
DOI: 10.1002/neu.20017

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Cellular bases of activity-dependent paralysis in Drosophila stress-sensitive mutants.PMID 15281071Read ↗

Mutations in dynamin-related protein result in gross changes in mitochondrial morphology and affect synaptic vesicle recycling at the Drosophila neuromuscular junction.Semantic proximity Read ↗

Neuropathology in Drosophila mutants with increased seizure susceptibility.Semantic proximity Read ↗

A temperature-sensitive allele of Drosophila sesB reveals acute functions for the mitochondrial adenine nucleotide translocase in synaptic transmission and dynamin regulation.Semantic proximity Read ↗

Novel mutations affecting the Na, K ATPase alpha model complex neurological diseases and implicate the sodium pump in increased longevity.Semantic proximity Read ↗

Neural dysfunction and neurodegeneration in Drosophila Na+/K+ ATPase alpha subunit mutants.Semantic proximity Read ↗

σ2-Adaptin Facilitates Basal Synaptic Transmission and Is Required for Regenerating Endo-Exo Cycling Pool Under High-Frequency Nerve Stimulation in Drosophila.Semantic proximity Read ↗

Genetic modifications of seizure susceptibility and expression by altered excitability in Drosophila Na(+) and K(+) channel mutants.Semantic proximity Read ↗

Loss of Activity-Induced Mitochondrial ATP Production Underlies the Synaptic Defects in a Drosophila Model of ALSSemantic proximity Read ↗

Shortened lifespan and other age-related defects in bang sensitive mutants of <i>Drosophila melanogaster</i>Semantic proximity Read ↗

Synaptic mitochondria are critical for mobilization of reserve pool vesicles at Drosophila neuromuscular junctions.Semantic proximity Read ↗

Phenotypic rescue of a Drosophila model of mitochondrial ANT1 disease.Semantic proximity Read ↗

Properties of short-term synaptic depression at larval neuromuscular synapses in wild-type and temperature-sensitive paralytic mutants of Drosophila.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.