Article
Reduced sodium appetite and increased oxytocin gene expression in mutant mice lacking beta-endorphin.
Neuroscience - 1 Jan 2003
Franchini L F, Rubinstein M, Vivas L
Abstract excerpt
Central opioid and oxytocinergic systems have been involved in the regulatory control of sodium appetite. In addition, previous studies support the existence of a functional interaction between opioid peptides and oxytocinergic pathways, and suggest that beta-endorphin neurons would modulate the activity of central oxytocinergic pathways, its pituitary secretion and sodium appetite. To investigate the role of...
Read the complete abstract on PubMedTopics
Share this publication in a Topic to start or enrich a Post.
