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Article

Animal cell mutants represent two complementation groups of peroxisome-defective Zellweger syndrome.

The Journal of clinical investigation - 1 Nov 1992

Shimozawa N, Tsukamoto T, Suzuki Y, Orii T, Fujiki Y

Abstract excerpt

Generalized peroxisome-deficient disorders including cerebro-hepato-renal Zellweger syndrome, neonatal adrenoleukodystrophy, and infantile Refsum disease are autosomal recessive diseases, where catalase-containing particles (peroxisomes) are morphologically absent. We previously isolated two Chinese hamster ovary (CHO) cell mutants (Z24 and Z65) that resemble the fibroblasts from patients with such diseases, in...

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Identifiers

PMID: 1430210
DOI: 10.1172/JCI116063
PMCID: PMC443247

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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Animal cell mutants represent two complementation groups of peroxisome-defective Zellweger syndrome.PMID 1430210Read ↗

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