Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Modulation of endotoxin-induced endothelial function by calcium/calmodulin-dependent protein kinase.

Shock (Augusta, Ga.) - 1 Aug 2003

Cuschieri Joseph, Gourlay David, Garcia Iris, Jelacic Sandra, Maier Ronald V

Abstract excerpt

Endothelial cells facilitate sepsis-induced neutrophil adherence through the production of adhesion molecules and proinflammatory cytokines. The production of these factors requires coordinated intracellular inflammatory signaling. Recently, patients prone to sepsis-induced complications have bee...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Calcium-Calmodulin-Dependent Protein Kinases
Share to Endotoxins
Share to Fluoresceins

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Intercellular Adhesion Molecule-1

Share to Mitogen-Activated Protein Kinase 1

Share to Mitogen-Activated Protein Kinase 3

Share to Transcription Factor AP-1

Share to Umbilical Veins

Source

Identifiers

PMID: 12865664
DOI: 10.1097/01.shk.0000074789.29800.a5

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 1 Sept 2026. Prepared record publication-12865664-2026-09-01.1. Manifest publication-graph-cohort-2026-09-01.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 1 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Modulation of endotoxin-induced endothelial function by calcium/calmodulin-dependent protein kinase.PMID 12865664Read ↗

ILK mediates LPS-induced vascular adhesion receptor expression and subsequent leucocyte trans-endothelial migration.Semantic proximity Read ↗

Angiopoietin-2 mediates thrombin-induced monocyte adhesion and endothelial permeability.Semantic proximity Read ↗

Effect of atorvastatin on adhesive phenotype of human endothelial cells activated by tumor necrosis factor alpha.Semantic proximity Read ↗

Selective activation of endothelial cells by the antioxidant pyrrolidine dithiocarbamate: involvement of C-jun N-terminal kinase and AP-1 activation.Semantic proximity Read ↗

Regulation of lung neutrophil recruitment by VE-cadherin.Semantic proximity Read ↗

Differential Regulation of CD44 Expression by Lipopolysaccharide (LPS) and TNF-α in Human Monocytic Cells: Distinct Involvement of c-Jun N-Terminal Kinase in LPS-Induced CD44 ExpressionSemantic proximity Read ↗

LPS-induced MCP-1 expression in human microvascular endothelial cells is mediated by the tyrosine kinase, Pyk2 via the p38 MAPK/NF-kappaB-dependent pathway.Semantic proximity Read ↗

Activation-induced depletion of protein kinase C alpha provokes desensitization of monocytes/macrophages in sepsis.Semantic proximity Read ↗

Regulation of G protein-coupled receptor activities by the platelet-endothelial cell adhesion molecule, PECAM-1.Semantic proximity Read ↗

A novel microfluidic assay reveals a key role for protein kinase C δ in regulating human neutrophil–endothelium interactionSemantic proximity Read ↗

Heterogeneous activation thresholds to cytokines in genetically distinct endothelial cells: evidence for diverse transcriptional responses.Semantic proximity Read ↗

Nitric oxide suppresses IL-8 transcription by inhibiting c-Jun N-terminal kinase-induced AP-1 activation.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.