Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Antioxidative enzymes in human nasal mucosa after exposure to ozone. Possible role of GSTM1 deficiency.

Inflammation research : official journal of the European Histamine Research Society ... [et al.] - 1 Feb 2003

Otto-Knapp R, Jurgovsky K, Schierhorn K, Kunkel G

Abstract excerpt

OBJECTIVE AND DESIGN: Epithelial antioxidative enzymes (AOEs) are thought to be a first line of defense against reactive oxygen species as they are upregulated after exposure to ozone according to animal studies. We analysed the activities of the AOEs catalase (CAT), glutathione peroxidase (GPX), glutathione reductase (GR), superoxide dismutase (SOD) and glutathione-S-transferase (GST) in a tissue culture of...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to CAT gene
Share to Genotype

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Glutathione Transferase

Share to Heterozygote

Share to Nasal Mucosa

Share to Oxidoreductases

Share to Polymorphism, Genetic

Source

Identifiers

PMID: 12665121
DOI: 10.1007/s000110300000

Genes

GSTM1
CAT

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Antioxidative enzymes in human nasal mucosa after exposure to ozone. Possible role of GSTM1 deficiency.PMID 12665121Read ↗

The glutathione-S-transferase Mu 1 null genotype modulates ozone-induced airway inflammation in human subjects.Semantic proximity Read ↗

Schoolchildren's antioxidation genotypes are susceptible factors for reduced lung function and airway inflammation caused by air pollution.Semantic proximity Read ↗

Biomarkers of oxidative stress after controlled human exposure to ozone.Semantic proximity Read ↗

Genetic polymorphism of GSTM1 and antioxidant supplementation influence lung function in relation to ozone exposure in asthmatic children in Mexico City.Semantic proximity Read ↗

Effects of antioxidant enzyme polymorphisms on ozone-induced lung function changes.Semantic proximity Read ↗

No evidence for a correlation of glutathione S-tranferase polymorphisms and chronic rhinosinusitis.Semantic proximity Read ↗

Genetic polymorphisms in antioxidative enzymes are associated to forced expiratory volume in 1 s (FEV1) in smokers independently of asthma.Semantic proximity Read ↗

GSTM1 and GSTP1 and respiratory health in asthmatic children exposed to ozone.Semantic proximity Read ↗

Ozone exposure, vitamin C intake, and genetic susceptibility of asthmatic children in Mexico City: a cohort study.Semantic proximity Read ↗

Environmental Pollution by Ozone, Oxidative Stress: the Relationship between the Intestine and Neurodegenerative DiseaseSemantic proximity Read ↗

Oxidative stress and genetic and epidemiologic determinants of oxidant injury in childhood asthma.Semantic proximity Read ↗

Ozone exposure, antioxidant genes, and lung function in an elderly cohort: VA normative aging study.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.