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Article

The expression of ETV6/CBFA2 (TEL/AML1) is not sufficient for the transformation of hematopoietic cell lines in vitro or the induction of hematologic disease in vivo

1 Oct 2001

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 11675129
DOI: 10.1016/s0165-4608(01)00518-0

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The expression of ETV6/CBFA2 (TEL/AML1) is not sufficient for the transformation of hematopoietic cell lines in vitro or the induction of hematologic disease in vivoPMID 11675129Read ↗

Defining the oncogenic function of the TEL/AML1 (ETV6/RUNX1) fusion protein in a mouse model.Semantic proximity Read ↗

Transformation of hematopoietic cell lines to growth‐factor independence and induction of a fatal myelo‐ and lymphoproliferative disease in mice by retrovirally transduced TEL/JAK2 fusion genesSemantic proximity Read ↗

The second ETV6 allele is not necessarily deleted in acute leukemias with a ETV6/ABL fusion.Semantic proximity Read ↗

The ETS factor TEL2 is a hematopoietic oncoproteinSemantic proximity Read ↗

AML1-Evi-1 specifically transforms hematopoietic stem cells through fusion of the entire Evi-1 sequence to AML1.Semantic proximity Read ↗

Mechanisms for the transformation of myeloid and erythroid cells by the nuclear oncogene V-myb.Semantic proximity Read ↗

TEL2, an ETS factor expressed in human leukemia, regulates monocytic differentiation of U937 Cells and blocks the inhibitory effect of TEL1 on ras-induced cellular transformation.Semantic proximity Read ↗

v-myb and v-ets transform chicken erythroid cells and cooperate both in trans and in cis to induce distinct differentiation phenotypes.Semantic proximity Read ↗

Avian models to study the transcriptional control of hematopoietic lineage commitment and to identify lineage-specific genes.Semantic proximity Read ↗

A functional Ets DNA-binding domain is required to maintain multipotency of hematopoietic progenitors transformed by Myb-Ets.Semantic proximity Read ↗

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