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Article

Bone development and inflammatory disease is regulated by AP-1 (Fos/Jun)

8 Dec 2009

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Share to Bone Metabolism and Diseases
Share to Cytokine Signaling Pathways and Interactions
Share to TGF-β signaling in diseases

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Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Identifiers

PMID: 19995753
DOI: 10.1136/ard.2009.119396

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Related research

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Research trail→

Click a neighbor to travelStep 1 · 12 closest

Bone development and inflammatory disease is regulated by AP-1 (Fos/Jun)PMID 19995753Read ↗

Functions of AP1 (Fos/Jun) in bone developmentSemantic proximity Read ↗

Signalling in osteoclasts and the role of Fos/AP1 proteinsSemantic proximity Read ↗

Activator protein 1 (Fos/Jun) functions in inflammatory bone and skin diseaseSemantic proximity Read ↗

Fosl1 is a transcriptional target of c-Fos during osteoclast differentiationSemantic proximity Read ↗

The transcription factor Fos: a Janus-type regulator in health and disease.Semantic proximity Read ↗

Minireview: Transcriptional Regulation in Development of BoneSemantic proximity Read ↗

Induction of the AP-1 members c-Jun and JunB by TGF-β/Smad suppresses early Smad-driven gene activationSemantic proximity Read ↗

Expression of the novel transcription factor OASIS, which belongs to the CREB/ATF family, in mouse embryo with special reference to bone developmentSemantic proximity Read ↗

Role and Regulation of Transcription Factors in OsteoclastogenesisSemantic proximity Read ↗

c-Fos: a Key Regulator of Osteoclast-Macrophage Lineage Determination and Bone RemodelingSemantic proximity Read ↗

Fra-2/AP-1 controls bone formation by regulating osteoblast differentiation and collagen production.Semantic proximity Read ↗

The Fos‐related antigen Fra‐1 is an activator of bone matrix formationSemantic proximity Read ↗

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