Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Oestrogen and progesterone increase the levels of apoptosis induced by the human papillomavirus type 16 E2 and E7 proteins

1 Jan 2001

Abstract excerpt

Human papillomavirus (HPV) type 16 infects the genital tract and is generally acknowledged to be a causative agent of cervical cancer. HPV infection alone is not sufficient to induce cervical cancer and other factors such as steroid hormones are thought to play a role in the establishment and/or progression of this disease. The HPV-16 E2 protein is required for virus replication and modulates viral gene...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cervical Cancer and HPV Research
Share to NF-κB Signaling Pathways
Share to Reproductive System and Pregnancy

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 11125173
DOI: 10.1099/0022-1317-82-1-201

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Oestrogen and progesterone increase the levels of apoptosis induced by the human papillomavirus type 16 E2 and E7 proteinsPMID 11125173Read ↗

The role of human papillomavirus oncoproteins E6 and E7 in apoptosisSemantic proximity Read ↗

Resveratrol inhibits the progression of cervical cancer by suppressing the transcription and expression of HPV E6 and E7 genesSemantic proximity Read ↗

Human papillomavirus oncoproteins and apoptosis (Review)Semantic proximity Read ↗

Elucidation of rutin’s role in inducing caspase-dependent apoptosis via HPV-E6 and E7 down-regulation in cervical cancer HeLa cellsSemantic proximity Read ↗

Transcription-independent triggering of the extrinsic pathway of apoptosis by human papillomavirus 18 E2 protein.Semantic proximity Read ↗

The E7 protein of human papillomavirus type 16 sensitizes primary human keratinocytes to apoptosisSemantic proximity Read ↗

Apoptosis in human papillomavirus16 E7-, but not E6-immortalized human uroepithelial cells.Semantic proximity Read ↗

Inhibition of cervical carcinoma cell line proliferation by the introduction of a bovine papillomavirus regulatory geneSemantic proximity Read ↗

Endogenous Human Papillomavirus E6 and E7 Proteins Differentially Regulate Proliferation, Senescence, and Apoptosis in HeLa Cervical Carcinoma CellsSemantic proximity Read ↗

P53 represses human papillomavirus type 16 DNA replication via the viral E2 protein.Semantic proximity Read ↗

Role of the retinoblastoma pathway in senescence triggered by repression of the human papillomavirus E7 protein in cervical carcinoma cells.Semantic proximity Read ↗

The human papillomavirus (HPV) 16 E2 protein induces apoptosis in the absence of other HPV proteins and via a p53-dependent pathway.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.