Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Leukemia relapse reconsidered from the molecular aspect.

Leukemia & lymphoma - 1 May 2000

Naoe T, Nakano Y, Kiyoi H

Abstract excerpt

Relapse, a major obstacle in the treatment of acute leukemia, is essentially caused by re-growth of residual leukemia cells, frequently accompanied by resistance to chemotherapy. Comparative studies of clones both at initial diagnosis and at subsequent relapse have indicated that phenotype and karyotype are frequently changed at relapse. This can be recognized as the result of negative selection by chemotherapy...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Clone Cells
Share to Immunophenotyping
Share to Karyotyping

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Leukemia

Share to Neoplasm, Residual

Share to Neoplastic Stem Cells

Share to Remission Induction

Share to Salvage Therapy

Source

Identifiers

PMID: 11042512
DOI: 10.3109/10428190009058504

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Leukemia relapse reconsidered from the molecular aspect.PMID 11042512Read ↗

AML evolution from preleukemia to leukemia and relapse.Semantic proximity Read ↗

Biology of post-transplant relapse: actionable features.Semantic proximity Read ↗

[Clinical and biological prognostic factors in relapsed acute myeloid leukemia patients].Semantic proximity Read ↗

Clonal evolution of acute myeloid leukemia from diagnosis to relapseSemantic proximity Read ↗

Molecular and genetic alterations associated with therapy resistance and relapse of acute myeloid leukemiaSemantic proximity Read ↗

Molecular evolution of acute myeloid leukaemia in relapse: unstable N-ras and FLT3 genes compared with p53 gene.Semantic proximity Read ↗

Late relapses evolve from slow-responding subclones in t(12;21)-positive acute lymphoblastic leukemia: evidence for the persistence of a preleukemic cloneSemantic proximity Read ↗

Tracing the origins of relapse in acute myeloid leukaemia to stem cells.Semantic proximity Read ↗

Proceedings from the National Cancer Institute's Second International Workshop on the Biology, Prevention, and Treatment of Relapse after Hematopoietic Stem Cell Transplantation: Part I. Biology of Relapse after TransplantationSemantic proximity Read ↗

Tracing relapse clone reveals complex recurrence patterns of pediatric acute lymphoblastic leukemiaSemantic proximity Read ↗

Relapse in children with acute lymphoblastic leukemia involving selection of a preexisting drug-resistant subcloneSemantic proximity Read ↗

Mutational landscape changes of AML in patients relapsing after allogeneic hematopoietic cell transplantation.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.