Article

NLGN3 contributes to angiogenesis in myocardial infarction via activation of the Gαi1/3-Akt Pathway

2024-10-27

Abstract excerpt

<h4>BACKGROUND</h4> Angiogenesis is an important repair mechanism for myocardial infarction. Neuroligin-3 (NLGN3) can promote angiogenesis by activating Gαi1/3-Akt signaling following ischemic brain injury. This study investigated the role of NLGN3 in myocardial infarction (MI). <h4>METHODS AND RESULTS</h4> On the 7 th day after MI, the plasma level of NLGN3 in patients was significantly higher than in the cont...

Topics

Open a Topic to create a Post that cites this publication.

Identifiers and source

Literature Corpus work: f7e48d37-6cd3-5c9c-b7ab-76fb997923e3
DOI: 10.1101/2024.10.24.620138

Open publication

Related research

Semantic proximity does not establish scientific evidence.

Click a neighbor to travelStep 1 · 12 closest

NLGN3 contributes to angiogenesis in myocardial infarction via activation of the Gαi1/3-Akt PathwayDOI 10.1101/2024.10.24.620138Read

Transcriptome analysis reveals the molecular mechanism of miR133a as a potential therapeutic target for acute myocardial infarction during angiogenesisSemantic proximity Read

The novel cardiokine GDF3 predicts adverse fibrotic remodeling post-myocardial infarctionSemantic proximity Read

Kindlin‐3 Promotes Angiogenesis via Notch Signalling and Is Crucial for Functional Recovery Postmyocardial InfarctionSemantic proximity Read

The whole transcriptome and proteome changes in the early stage of myocardial infarctionSemantic proximity Read

Fibulin-3 is necessary to prevent cardiac rupture following myocardial infarctionSemantic proximity Read

Ablation of SGK1 impairs endothelial cell migration and tube formation leading to decreased neo-angiogenesis following myocardial infarction.Semantic proximity Read

G protein subunit alpha i2's pivotal role in angiogenesisSemantic proximity Read

RNF213 Knockdown and Inflammation Promote Angiogenesis and Phenotype Switching by JAK2/STAT3 Pathway in Moyamoya DiseaseSemantic proximity Read

lncRNA-MIAT Regulates Microvascular Dysfunction by Functioning as a Competing Endogenous RNASemantic proximity Read

Angiogenesis after acute myocardial infarction.Semantic proximity Read

NR3C2 activates LCN2 transcription to promote endoplasmic reticulum stress and cell apoptosis in ischemic cerebral infarctionSemantic proximity Read

The lncRNA Malat1 regulates microvascular function after myocardial infarction in mice via miR-26b-5p/Mfn1 axis-mediated mitochondrial dynamicsSemantic proximity Read