Article

Intermittent Hypoxia Alters Cerebrovascular Recovery After Stroke

2026-01-14

Abstract excerpt

Stroke is a prevalent chronic disease, significantly contributing to mortality and long-term disability. The cerebrovascular recovery process after stroke can be complicated by certain comorbidities, including obstructive sleep apnea syndrome (OSA). However, the mechanisms underlying this deleterious impact of OSA on post-stroke recovery remain elusive. We conducted a preclinical study in rats submitted to stroke...

Topics

Open a Topic to create a Post that cites this publication.

Identifiers and source

Literature Corpus work: 54a70762-366d-5d18-b0a5-8e96a5b43433
DOI: 10.64898/2026.01.11.698922

Open publication

Related research

Semantic proximity does not establish scientific evidence.

Click a neighbor to travelStep 1 · 12 closest

Intermittent Hypoxia Alters Cerebrovascular Recovery After StrokeDOI 10.64898/2026.01.11.698922Read

Cerebral hypoperfusion exacerbates vascular dysfunction after traumatic brain injurySemantic proximity Read

Animal models of ischemic cerebral small vessel disease: A narrative review of pathological phenotypes, challenges, and future directions.Semantic proximity Read

Brain Injury: Response to Injury Wound Healing Mechanisms and Enlarged Perivascular Spaces in Obesity, Metabolic Syndrome, and Type 2 Diabetes MellitusSemantic proximity Read

Vascular neural network phenotypic transformation after traumatic injury: potential role in long-term sequelae.Semantic proximity Read

Moyamoya Disease: Physiological Mechanisms and Treatment ApproachesSemantic proximity Read

The Impact of Environmental Enrichment on Neurological Damage: Mechanistic Study of Ferroptosis Inhibition via Tlr4Semantic proximity Read

Neuropathological hallmarks during the chronic phase of ischemic stroke in mice and humansSemantic proximity Read

The Potential Role of Hypoxia-Inducible Factor-1 in the Progression and Therapy of Central Nervous System DiseasesSemantic proximity Read

Endothelial microvesicles induce blood-brain barrier and neuronal damage caused by intermittent hypoxia through Fas/FasL-caspase8 signaling pathwaySemantic proximity Read

Collaterals influence oxygen metabolism on admission MRI in acute ischemic strokeSemantic proximity Read

Differential S1PR Expression and Altered Barrier, and Inflammatory Marker Levels in Human Brain Endothelial Cells Following Acute Ischemia-Reperfusion Like InjurySemantic proximity Read

Protective effect of carotid endarterectomy: inducing ischemic tolerance in brain tissue after strokeSemantic proximity Read